Deletion Polymorphism of Disc1 Is Common to All 129 Mouse Substrains: Implications for Gene-Targeting Studies of Brain Function Export

@article{citeulike:815319, abstract = {We report that the Disc1 gene in all extant 129 mouse inbred substrains has a deletion, previously considered specific to the 129S6/SvEv substrain, which is predicted to abolish production of the full-length protein. This finding has implications for the study of knockout mice generated from 129-derived embryonic stem cells. 10.1534/genetics.106.060749}, author = {Clapcote, Steven J. and Roder, John C.}, citeulike-article-id = {815319}, citeulike-linkout-0 = {http://dx.doi.org/10.1534/genetics.106.060749}, citeulike-linkout-1 = {http://www.genetics.org/cgi/content/abstract/173/4/2407}, citeulike-linkout-2 = {http://view.ncbi.nlm.nih.gov/pubmed/16751659}, citeulike-linkout-3 = {http://www.hubmed.org/display.cgi?uids=16751659}, day = {1}, doi = {10.1534/genetics.106.060749}, journal = {Genetics}, keywords = {129, disc1, mir, mouse-ko}, month = {August}, number = {4}, pages = {2407--2410}, posted-at = {2006-08-24 09:23:34}, priority = {2}, title = {Deletion Polymorphism of Disc1 Is Common to All 129 Mouse Substrains: Implications for Gene-Targeting Studies of Brain Function}, url = {http://dx.doi.org/10.1534/genetics.106.060749}, volume = {173}, year = {2006} }

TY - JOUR ID - citeulike:815319 L3 - citeulike-article-id:815319 N2 - We report that the Disc1 gene in all extant 129 mouse inbred substrains has a deletion, previously considered specific to the 129S6/SvEv substrain, which is predicted to abolish production of the full-length protein. This finding has implications for the study of knockout mice generated from 129-derived embryonic stem cells. 10.1534/genetics.106.060749 IS - 4 JF - Genetics EP - 2410 TI - Deletion Polymorphism of Disc1 Is Common to All 129 Mouse Substrains: Implications for Gene-Targeting Studies of Brain Function VL - 173 SP - 2407 KW - 129 KW - disc1 KW - mir KW - mouse-ko AU - Clapcote, Steven AU - Roder, John PY - 2006/08/01/ UR - http://dx.doi.org/10.1534/genetics.106.060749 ER -