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<pubDate>Thu, 07 Aug 2008 21:41:51 BST</pubDate>


	<title>CiteULike: Jozefs's library [3 articles]</title>
	<description>CiteULike: Jozefs's library [3 articles]</description>


	<link>http://www.citeulike.org/user/Jozefs</link>
	<dc:publisher>CiteULike.org</dc:publisher>
	<dc:language>en-gb</dc:language>
	<dc:rights>Copyright &#169; 2004-2008 citeulike.org</dc:rights>
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        <rdf:li rdf:resource="http://www.citeulike.org/user/Jozefs/article/2365212"/>
        <rdf:li rdf:resource="http://www.citeulike.org/user/Jozefs/article/2361703"/>
        <rdf:li rdf:resource="http://www.citeulike.org/user/Jozefs/article/2238389"/>

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<item rdf:about="http://www.citeulike.org/user/Jozefs/article/2365212">
    <title>Genetic investigation of modern burned corpses</title>
    <link>http://www.citeulike.org/user/Jozefs/article/2365212</link>
    <description>&lt;i&gt;International Congress Series, Vol. 1261 (April 2004), pp. 50-52.&lt;/i&gt;&lt;br /&gt;&lt;br /&gt;Burning of corpses is a well-known and widely spread funeral procedure that has been performed for a long time in many cultures. Nowadays more and more corpses are burned in cremations and buried in urns, often for practical and financial reasons. Usually cremation takes place as temperatures of over 1000 [degree sign]C for more than 70 min, destroying the corpse and only leaving severely burned teeth and some fragments of larger bones. In some scientific, criminal or civil cases even after cremation, there is the need of genetic investigations for identification or paternity testing. Here we present a systematic genetic investigation of 10 corpses that were burned in a crematory. After DNA extraction of the remains, the presence of human nuclear and mitochondrial DNA was tested by a highly sensitive Duplex PCR and quantified via real time PCR; genetic typing was done using the AmpFlSTR Identifiler kit.</description>
    <dc:title>Genetic investigation of modern burned corpses</dc:title>

    <dc:creator>N von Wurmb-Schwark</dc:creator>
    <dc:creator>E Simeoni</dc:creator>
    <dc:creator>A Ringleb</dc:creator>
    <dc:creator>M Oehmichen</dc:creator>
    <dc:identifier>doi:10.1016/S0531-5131(03)01830-2</dc:identifier>
    <dc:source>International Congress Series, Vol. 1261 (April 2004), pp. 50-52.</dc:source>
    <dc:date>2008-02-12T08:33:31-00:00</dc:date>
    <prism:publicationYear>2004</prism:publicationYear>
    <prism:publicationName>International Congress Series</prism:publicationName>
    <prism:volume>1261</prism:volume>
    <prism:startingPage>50</prism:startingPage>
    <prism:endingPage>52</prism:endingPage>
    <prism:category>crematie</prism:category>
    <prism:category>lijken</prism:category>
    <prism:category>verbranding</prism:category>
</item>



<item rdf:about="http://www.citeulike.org/user/Jozefs/article/2361703">
    <title>Alcohol Consumption and the Risk of Hypertension in Women and Men</title>
    <link>http://www.citeulike.org/user/Jozefs/article/2361703</link>
    <description>&lt;i&gt;Hypertension (7 February 2008), HYPERTENSIONAHA.107.104968.&lt;/i&gt;&lt;br /&gt;&lt;br /&gt;AbstractHeavy alcohol intake increases the risk of hypertension, but the relationship between light-to-moderate alcohol consumption and incident hypertension remains controversial. We prospectively followed 28 848 women from the Women's Health Study and 13 455 men from the Physicians' Health Study free of baseline hypertension, cardiovascular disease, and cancer. Self-reported lifestyle and clinical risk factors were collected. In women, total alcohol intake was summed from liquor, red wine, white wine, and beer; men reported total alcohol intake from a single combined question. During 10.9 and 21.8 years of follow-up, 8680 women and 6012 men developed hypertension (defined as new physician diagnosis, antihypertensive treatment, reported systolic blood pressure [&#8805;]140 mm Hg, or diastolic blood pressure [&#8805;]90 mm Hg). In women, we found a J-shaped association between alcohol intake and hypertension in age- and lifestyle-adjusted models. Adding potential intermediates (body mass index, diabetes, and high cholesterol) attenuated the benefits of alcohol in the light-to-moderate range and strengthened the adverse effects of heavy alcohol intake. Beverage-specific relative risks paralleled those for total alcohol intake. In men, alcohol intake was positively and significantly associated with the risk of hypertension and persisted after multivariate adjustment. Models stratified by baseline systolic blood pressure (&#60;120 versus [&#8805;]120 mm Hg) or diastolic blood pressure (&#60;75 versus [&#8805;]75 mm Hg) did not alter the relative risks in women and men. In conclusion, light-to-moderate alcohol consumption decreased hypertension risk in women and increased risk in men. The threshold above which alcohol became deleterious for hypertension risk emerged at [&#8805;]4 drinks per day in women versus a moderate level of [&#8805;]1 drink per day in men. 10.1161/HYPERTENSIONAHA.107.104968</description>
    <dc:title>Alcohol Consumption and the Risk of Hypertension in Women and Men</dc:title>

    <dc:creator>Howard Sesso</dc:creator>
    <dc:creator>Nancy Cook</dc:creator>
    <dc:creator>Julie Buring</dc:creator>
    <dc:creator>Joann Manson</dc:creator>
    <dc:creator>Michael Gaziano</dc:creator>
    <dc:identifier>doi:10.1161/HYPERTENSIONAHA.107.104968</dc:identifier>
    <dc:source>Hypertension (7 February 2008), HYPERTENSIONAHA.107.104968.</dc:source>
    <dc:date>2008-02-11T08:24:07-00:00</dc:date>
    <prism:publicationYear>2008</prism:publicationYear>
    <prism:publicationName>Hypertension</prism:publicationName>
    <prism:startingPage>HYPERTENSIONAHA.107.104968</prism:startingPage>
    <prism:category>alcohol</prism:category>
    <prism:category>bloeddruk</prism:category>
    <prism:category>wijn</prism:category>
</item>



<item rdf:about="http://www.citeulike.org/user/Jozefs/article/2238389">
    <title>Marketing actions can modulate neural representations of experienced pleasantness</title>
    <link>http://www.citeulike.org/user/Jozefs/article/2238389</link>
    <description>&lt;i&gt;Proceedings of the National Academy of Sciences (14 January 2008), 0706929105.&lt;/i&gt;&lt;br /&gt;&lt;br /&gt;Despite the importance and pervasiveness of marketing, almost nothing is known about the neural mechanisms through which it affects decisions made by individuals. We propose that marketing actions, such as changes in the price of a product, can affect neural representations of experienced pleasantness. We tested this hypothesis by scanning human subjects using functional MRI while they tasted wines that, contrary to reality, they believed to be different and sold at different prices. Our results show that increasing the price of a wine increases subjective reports of flavor pleasantness as well as blood-oxygen-level-dependent activity in medial orbitofrontal cortex, an area that is widely thought to encode for experienced pleasantness during experiential tasks. The paper provides evidence for the ability of marketing actions to modulate neural correlates of experienced pleasantness and for the mechanisms through which the effect operates. 10.1073/pnas.0706929105</description>
    <dc:title>Marketing actions can modulate neural representations of experienced pleasantness</dc:title>

    <dc:creator>Hilke Plassmann</dc:creator>
    <dc:creator>John O'Doherty</dc:creator>
    <dc:creator>Baba Shiv</dc:creator>
    <dc:creator>Antonio Rangel</dc:creator>
    <dc:identifier>doi:10.1073/pnas.0706929105</dc:identifier>
    <dc:source>Proceedings of the National Academy of Sciences (14 January 2008), 0706929105.</dc:source>
    <dc:date>2008-01-16T09:30:58-00:00</dc:date>
    <prism:publicationYear>2008</prism:publicationYear>
    <prism:publicationName>Proceedings of the National Academy of Sciences</prism:publicationName>
    <prism:startingPage>0706929105</prism:startingPage>
    <prism:category>psychologie</prism:category>
    <prism:category>wijn</prism:category>
</item>



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