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✔ Sulforaphane and alpha-lipoic acid upregulate the expression of the pi class of glutathione S-transferase through c-jun and Nrf2 activation.

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The Journal of nutrition, Vol. 140, No. 5. (May 2010), pp. 885-892, doi:10.3945/jn.110.121418
by Chong-Kuei K. Lii, Kai-Li L. Liu, Yi-Ping P. Cheng, Ai-Hsuan H. Lin, Haw-Wen W. Chen, Chia-Wen W. Tsai
posted to no-tag by HEIRS ✚ on 2010-04-27 16:01:53 *** along with 1 group HEIRS - Environmental Illness Research
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Abstract

The anticarcinogenic effect of dietary organosulfur compounds has been partly attributed to their modulation of the activity and expression of phase II detoxification enzymes. Our previous studies indicated that garlic allyl sulfides upregulate the expression of the pi class of glutathione S-transferase (GSTP) through the activator protein-1 pathway. Here, we examined the modulatory effect of sulforaphane (SFN) and alpha-lipoic acid (LA) or dihydrolipoic acid (DHLA) on GSTP expression in rat Clone 9 liver cells. Cells were treated with LA or DHLA ...
 

THE EFFECTS OF PSYCHOLOGICAL STRESS ON HUMANS: INCREASED PRODUCTION OF PRO-INFLAMMATORY CYTOKINES AND Th1-LIKE RESPONSE IN STRESS-INDUCED ANXIETY

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Cytokine, Vol. 10, No. 4. (April 1998), pp. 313-318
by Michael Maes, Cai Song, Aihua Lin, et al.Raf De Jongh, An Van Gastela, Gunter Kenis, Eugene Bosmans, Ingrid De Meester, Ina B. Neels, Paul Demedts, Aleksandar Janca, Simon Scharpé, Ronald S. Smith
posted to anxiety cytokines inflammation stress th1 by HEIRS ✚ on 2009-03-08 01:46:08 *** along with 2 people and 4 groups diogocandeo mtf5024 HEIRS - Environmental Illness Research HEIRS Cellular Biology HEIRS Immunology and Autoimmune Diseases HEIRS Psychology and Psychotherapy
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Elevated serum interleukin-6 (IL-6) and IL-6 receptor concentrations in posttraumatic stress disorder following accidental man-made traumatic events

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Biological Psychiatry, Vol. 45, No. 7. (1999), pp. 833-839
by M. Maes, A. H. Lin, L. Delmeire, et al. Van, G. Kenis, De, E. Bosmans
posted to interleukin-6 ptsd by HEIRS ✚ on 2008-07-30 18:32:18 *** along with 2 groups HEIRS - Environmental Illness Research HEIRS PTSD
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Background: Recently, it has been reported that serum interleukin-1β (IL-1β), but not soluble IL-2 receptor (slL-2R), concentrations were significantly higher in patients with posttraumatic stress disorder (PTSD) than in normal volunteers, and that psychological stress in humans is associated with increased secretion of proinflammatory cytokines, such as IL-6. Methods: The aim of the present study was to examine the inflammatory response system in patients with PTSD through measurements of serum IL-6, sIL-6R, sgp130 (the IL-6 signal transducing protein), sIL-1R antagonist (slL-1RA; ...
 

Deficiency in neuronal TGF-β signaling promotes neurodegeneration and Alzheimer’s pathology

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The Journal of Clinical Investigation, Vol. 116, No. 11. (2006), pp. 3060-3069
by Ina Tesseur, Kun Zou, Luke Esposito, et al.Frederique Bard, Elisabeth Berber, Judith Van Can, Amy H. Lin, Leslie Crews, Patrick Tremblay, Paul Mathews, Lennart Mucke, Eliezer Masliah, Tony Wyss-Coray
posted to alzheimers neurodegeneration tgf-1 by HEIRS ✚ on 2008-06-29 23:21:27 ** along with 1 group HEIRS - Environmental Illness Research
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Abstract

Alzheimer’s disease (AD) is characterized by progressive neurodegeneration and cerebral accumulation of the β-amyloid peptide (Aβ), but it is unknown what makes neurons susceptible to degeneration. We report that the TGF-β type II receptor (TβRII) is mainly expressed by neurons, and that TβRII levels are reduced in human AD brain and correlate with pathological hallmarks of the disease. Reducing neuronal TGF-β signaling in mice resulted in age-dependent neurodegeneration and promoted Aβ accumulation and dendritic loss in a mouse model of AD. ...
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