HEIRS's Matsuya:Y [1 article]

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Treatment with SRT1720, a SIRT1 Activator, Ameliorates Fatty Liver with Reduced Expression of Lipogenic Enzymes in MSG Mice.

American journal of physiology. Endocrinology and metabolism (1 September 2009)

by Yu Yamazaki, Isao Usui, Yukiko Kanatani, et al.Yuji Matsuya, Koichi Tsuneyama, Shiho Fujisaka, Agussalim Bukhari, Hikari Suzuki, Satoko Senda, Shingo Imanishi, Kazuya Hirata, Manabu Ishiki, Ryuji Hayashi, Masaharu Urakaze, Hideo Nemoto, Masashi Kobayashi, Kazuyuki Tobe

posted to no-tag by HEIRS on 2009-09-15 23:01:42

along with 1 group HEIRS - Environmental Illness Research

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Abstract

Nonalcoholic fatty liver disease (NAFLD) is an abnormal liver metabolism often observed with insulin resistance and metabolic syndrome. Calorie restriction is a useful treatment for NAFLD and reportedly prolongs the life spans of several species in which sirtuin plays an important role. In this study, we examined whether the activation of SIRT1, a mammalian ortholog of sirtuin, may ameliorate the development of NAFLD. MSG mice, which exhibited obesity and insulin resistance, were treated with SRT1720, a specific SIRT1 activator from the ...

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HEIRS's Matsuya:Y [1 article]

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