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Nat Neurosci, Vol. 16, No. 2. (06 February 2013), pp. 183-192, doi:10.1038/nn.3295 Key: citeulike:11975778
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Treatment of pain with morphine leads to paradoxical hyperalgesia. The authors provide evidence that morphine-induced hyperalgesia is a result of downregulation of the chloride transporter KCC2 in spinal lamina I neurons. Microglial expression of P2X4 receptors and release of BDNF may underlie this change in neuronal chloride homeostasis and morphine-induced hyperalgesia.
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