FANCF methylation contributes to chemoselectivity in ovarian cancer. Export

@article{citeulike:3451002, abstract = {A new model of ovarian cancer tumor progression implicates aberrant FANCF promoter methylation that is associated with gene silencing and disruption of the Fanconi-anemia-BRCA pathway. Disruption of the pathway occurs de novo in ovarian cancers and may contribute to selective sensitivity to platinum salts.}, author = {Olopade, O. I. and Wei, M.}, citeulike-article-id = {3451002}, citeulike-linkout-0 = {http://view.ncbi.nlm.nih.gov/pubmed/12781358}, citeulike-linkout-1 = {http://www.hubmed.org/display.cgi?uids=12781358}, issn = {1535-6108}, journal = {Cancer cell}, keywords = {brca, fa, ovarian}, month = {May}, number = {5}, pages = {417--420}, posted-at = {2008-10-26 01:41:59}, priority = {2}, title = {FANCF methylation contributes to chemoselectivity in ovarian cancer.}, url = {http://view.ncbi.nlm.nih.gov/pubmed/12781358}, volume = {3}, year = {2003} }

TY - JOUR ID - citeulike:3451002 L3 - citeulike-article-id:3451002 N2 - A new model of ovarian cancer tumor progression implicates aberrant FANCF promoter methylation that is associated with gene silencing and disruption of the Fanconi-anemia-BRCA pathway. Disruption of the pathway occurs de novo in ovarian cancers and may contribute to selective sensitivity to platinum salts. IS - 5 JF - Cancer cell SN - 1535-6108 EP - 420 TI - FANCF methylation contributes to chemoselectivity in ovarian cancer. VL - 3 SP - 417 KW - brca KW - fa KW - ovarian AU - Olopade, OI AU - Wei, M PY - 2003/05// UR - http://view.ncbi.nlm.nih.gov/pubmed/12781358 ER -